A Japanese research group published a communiqué on the 27th that they found that when the experimental rats suffered from myocardial infarction, activation of a protein on the surface of the experimental rats' myocardial cells could inhibit the necrosis of the myocardial cells.
This protein was discovered by a scientific research group of the Japan Natural Science Research Institute and is called "CFTR ion channel". When this protein is activated, the chloride ions in the cells are released to prevent the cells from swelling and necrosis.
The researchers found that when the experimental rats had myocardial infarction, if they were quickly injected with drugs that can activate the "CFTR ion channel", the myocardial cell necrosis rate was 5% to 10%, while about 40% of the experimental mice without injection of activation drugs Myocardial cell necrosis. In addition, half of the cardiomyocytes were necrotic in the experimental rats injected with drugs that blocked this protein activity.
This result is expected to help find new treatments for myocardial infarction. The head of this study, Okada Takashi, said: "If humans are also injected with drugs that can activate the 'CFTR ion channel', it should also be able to curb myocardial cell necrosis." Related papers have been Published in the online version of the journal "Cell Physiology and Biochemistry".
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